Et al., 2008), Arabidopsis (Lee et al., 2009), and tomato (Leide et al.
Et al., 2008), Arabidopsis (Lee et al., 2009), and tomato (Leide et al., 2011). Furthermore, Suttle et al. (2013) showed that endogenous ABA concentrations in potato tubers lower just after injury and attain a minimum soon after 24 h; nonetheless, the concentration then increases in the third for the seventh day in a pattern parallel to that of FHT (Fig. 7A). Furthermore, Lulai et al. (2008) reported that endogenous ABA concentrations improve after tuber harvest after which lower for the duration of tuber storage, displaying an age-dependent pattern also comparable to that of FHT (Fig. 5). According to Kumar et al. (2010), therapy with ABA partly restores the healing ability of older tubers by enhancing the accumulation of suberin aromatics. These authors also demonstrated that the age-induced loss of the healing capability is partly as a result of a reduced capacity to accumulate ABA and modulate the production of suberin Nav1.2 web aromatics via PAL. A related modulation could also be contemplated via FHT. However, injury of potato tubers triggers a speedy improve (by 5-fold) on the basal JA content which peaks four h after wounding and thereafter returns to basal levels, a pattern compatible having a role in the early wound response (Koda and Kikuta, 1994). However, Lulai et al. (2011) showed no effect of JA treatment or inhibition of JA accumulation on suberin biosynthesis within the wound closing layer, in agreement using the lack of an enhancing or inhibiting effect of JA with regard to FHT induction (Fig. 8B). In contrast, Ozeretskovskaya et al. (2009) reported a optimistic impact of exogenous JA in reference to periderm proliferation, but this acquiring opposes the more common view that one of several functions on the wound-induced JA is associated with the inhibition of growth by mitotic suppression (Zhang et al., 2008). Concerning SA, its function in wound responses hasFHT is induced by TrkA drug injuryTissues react to injury by forming a suberized and lignified closing layer which in most tissues is followed by active cell division that offers rise to a new phellogen and thereafter a wound periderm. In potato, leaves are characterized by the formation of a closing layer that is adjacent for the wounded margin and lacks cell division (Bloch, 1941), though tubers create a wound periderm as has been extensively documented (see, among other people, Morris et al., 1989; Sabba and Lulai, 2002). In leaves, FHT protein accumulation peaks following the third day following wounding when the formation in the closing layer is completed (Fig. 6A). In tubers, FHT accumulates early but keeps increasing at least up to the sixth day soon after injury (Fig. 7A) when the formation of the wound periderm is almost completed. These observations prove a fast and massive induction of FHT during the healing process concomitant with suberin deposition. It has been shown that deposition on the aromatic suberin precedes that from the aliphatic suberin (Yang and Bernards, 2006). In mechanically injured potato leaves, the gene encoding phenylalanine ammonia lyase (PAL), an enzyme that operates in the very3234 | Boher et far not been elucidated (Vlot et al., 2009). Earlier experiments making use of potato discs have to date been unable to detect any impact of exogenous SA in connection using the healing process (Ozeretskovskaya et al., 2009). Nonetheless, SA impedes FHT induction soon after injury (Fig. 8C), acting in an antagonistic manner with respect to ABA. The antagonistic interaction among the ABA and SA signalling pathways has already been r.